Chapter
9: The Basic Food Groups, or Much of What You Have
Been Taught About Diet is Probably Wrong /
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In Chapter 1 we
discussed generally how diabetics and nondiabetics
might react to a particular meal. Here we'll talk
about how the specific kinds of foods can affect your
blood sugar.
Perhaps the most curious
fact about diet, nutrition, and medication is that
while we can make accurate generalizations about how
most of us will react to a particular diet or medical
regimen, each individual will react somewhat differently
to a given food.
The foods we consume, once
you take away the water and undigestible contents,
can be grouped into three major categories: protein,
carbohydrate, and fat. Seldom is food from one of
these major groups solely one type of nutrient. Protein
foods often contain fat; carbohydrate foods frequently
contain some protein and fat. The only foods that
are virtually 100 percent fat are oils, butter, and
margarine.
Since our principal concern
here is blood sugar control, we'll concentrate on
how the three major types of nutrients affect blood
sugar. If you're a long-standing diabetic and have
followed the standard ADA diet for years, you'll find
that much of what you're about to read is radically
at odds with the ADA's dietary guidelines—and with
good reason, as you'll soon learn.
When we eat, the digestive
process breaks down the three major food groups into
their building blocks. These building blocks are then
absorbed into the bloodstream and reassembled into
the various products our bodies need in order to function.
Protein
Proteins are chains
of building blocks called amino acids. Through digestion,
dietary proteins are broken down by enzymes in the
digestive tract into their amino acid components.
These amino acids can then be reassembled not only
into muscle, nerve fiber, and vital organs, but also
into hormones, enzymes, and neurochemicals.
We acquire dietary protein
from many sources, but the foods that are richest
in it are egg whites, cheese, and meat (including
fish and fowl). Protein is available in much lower
amounts from vegetable sources such as legumes (beans),
seeds, and nuts, which also contain the other nutrients,
fat and carbohydrate.*
As stunning as it sounds—and
unbelievable, given the popular media's recent love
affair with a high "complex" carbohydrate,
low-fat diet—you can quite easily survive on a diet
in which you would eat no carbohydrate. Furthermore,
by sticking to a diet that contains no carbohydrate,
but high levels of fat and protein, you can reduce
your cardiac risk profile—serum cholesterol, blood
lipids, et cetera—though you'd deprive yourself of
all the "fun foods" that we crave most.
We've all been trained to think that carbohydrates
are our best, most benign source of food, so how can
this be?
Protein is the second of
our two dietary sources of blood sugar. Protein foods
are only about 20 percent protein by weight (6 grams
per ounce), the rest being fat, water, or undigestible
"gristle." The liver, instructed by the
hormone glucagon, can very slowly transform as much
as 52 percent of the above 6 grams per ounce into
glucose† if blood sugar descends too low or the body's
other amino acid needs have been met. Neither carbohydrate
nor fat can be transformed into protein.
In the 1920s, Arctic explorer
Vilhjalmur Stefansson noted in his travels that Eskimos
seemed to fare quite nicely on a zero-carbohydrate
diet (the Arctic being not exactly the ideal climate
for cultivating fruits, grain, or vegetables). Under
the watchful eyes of physicians from New York's Cornell
University Medical College and Bellevue Hospital,
Stefansson and a colleague submitted themselves to
a meat-only diet for a year. They ate 2,500 calories
a day, of which 75 percent was fat. As reported in
the Journal of the American Medical Association, on
July 6, 1929, the two men finished out their year
of a no-carbohydrate diet not only slimmer—each had
lost 6 pounds—but with reduced (and completely normal)
cholesterol. It's worth repeating that the men were
eating a diet with 75 percent of the calories coming
from fat. Current recommendations are to eat no more
than 30 percent of calories as fat—which very few
people can maintain—and there are some recommendations
for even lower percentages than that.
This is almost precisely
the opposite of the prevailing "wisdom,"
which says that if you want to lose weight and get
your cholesterol down, you need to eat lots of fruit,
vegetables, and grain products, and cut out meat as
much as possible. Despite this prevailing "wisdom,"
many contemporary dietary researchers exploring this
phenomenon have begun to arrive at the conclusion
that a high-carbohydrate diet is not so benign. In
fact, it has been shown—and it is my own observation—that
such a diet can increase body weight, increase blood
insulin levels, and raise most cardiac risk factors.
Why?
The answer is really quite
simple. The advent of our agricultural society is
comparatively recent in evolutionary terms—that is,
it began only about 10,000 years ago. For the millions
of years that preceded the constant availability of
grain and vegetable products, our ancestors were hunter-gatherers,
and ate what was available to them in the immediate
environment, primarily meat, fish, nuts—food that
was present year-round, and predominantly protein
and fat. In the summers they may have eaten fruits
and berries that were available locally in some regions,
but if they stored away fat during those seasons,
that fat was quickly burned up during the winter.
Although for the past two centuries, fruit, grain,
and vegetables have, in one form or another, been
available to us in this country year-round, our collective
food supply has historically been interrupted often
by famine—in some cultures more than others. The history
of the planet as best as we can determine is one of
feast and famine, and suggests that famine will strike
again and again. The recent famines in Ethiopia and
Somalia are examples.
Curiously, the genetic
predisposition toward obesity, or what today seems
in our society to be a predisposition toward obesity,
functioned during the famines of prehistory as an
effective method of survival. Ironically, the ancestors
of those who today are most at risk for Type II diabetes
were, during prehistory, not the sick and dying, but
the survivors. If famine struck today in the United
States, guess who would survive most easily? The same
people who are most at risk for Type II diabetes.
You can take this knowledge
and make it work for you rather than against you.
If you give it some thought, it makes perfect sense:
If a farmer wants to fatten up his pigs or cows, he
doesn't feed them meat or butter and eggs, he feeds
them grain. If you want to fatten yourself up, just
start loading up on bread, pasta, potatoes, cake,
and cookies—all high-carbohydrate foods. If you are
already obese, you know and I know that you crave—and
consume—these foods and probably avoid fats.
In many respects—and going
against the grain of a number of the medical establishment's
accepted notions about diabetics and protein—protein
will become the most important part of your diet if
you are going to control blood sugars.
If you
are a long-standing diabetic and are frustrated with
the care you've received over the years, you have
probably been conditioned to think that protein is
more of a poison than sugar and is the cause of kidney
disease. I was conditioned the same way—many years
ago, as I mentioned, I had laboratory evidence of
advanced proteinuria, signifying potentially fatal
kidney disease—but in this case, the conventional
wisdom is just a myth.
Nondiabetics who eat a
lot of protein don't get diabetic kidney disease.
Diabetics with normalized blood sugars don't get diabetic
kidney disease. High levels of dietary protein do
not cause kidney disease in diabetics or anyone else.
There is no higher incidence of kidney disease in
the cattle-growing states of the United States, where
many people eat steak every day, than there is in
the states where beef is more expensive and consumed
to a much lesser degree. Similarly, the incidence
of kidney disease in vegetarians is the same as the
incidence of kidney disease in nonvegetarians. It
is the high blood sugar levels that are unique to
diabetes, and to a much lesser degree the high levels
of insulin required to cover them (causing hypertension),
that cause the complications associated with diabetes.
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